B) Many animals are captured or killed by poachers, significantly reducing their numbers.
C) Viral epidemics that break out in human populations spread to animals in the ecosystem.
D) Factories cause a significant rise in temperature in the forests, endangering many species.
Answer: A) Urbanization leads to deforestation and loss of habitat for many species.
Explanation:
Urbanization is the main reason for the exploitation of the rain forests which has lead to the extinction of species used to be residing in the forests ecosystems. Following are the factors which are responsible for such disaster with the expension of urbanization:
1. Infrastructure: The land is the main requirement which is responsible for the exploitation of rain forests. The large area is required for the accommodation of the large human population, construction of roads, highways and bridges. This has replaced many habit-ant animals from their native land and also leads to extinction due to ecological disturbance.
2. Increasing needs of food, herbs and wood: Many species of wooden trees could be found in the rain forests which have now extincted due to increasing demand for wood. The herbs have been wiped up for the preparation of medicines and drugs. The large number of tree populations are also exploited due to availability of fruits, and vegetables.
3. Wildlife trafficking and hunting: Some of the animal species have been included in the list of endangered species due to their use for commercial selling of horns, hives and other valuables.
Answer: An extra cytoplasm enhance survival of a developing oocyte in such a way because cytoplasm serves as a reservoir of lipids, polysaccharide and lipids for the developing embryo.The more the cytoplasm content, more will be the nutrients for the developing embryo. The cytoplasm forms the yolk and provides nourishment to the developing embryo. As we know that the developing embryo requires more nutrition and the main source of nutrition is cytoplasm because the sperm has no reservoir of its own to provide nutrition to embryo.
Thus, an extra cytoplasm enhance the rate of survival of a developing oocyte.
Extra cytoplasm in a developing oocyte supports survival by providing necessary nutrients from fertilization to implantation, and energy for cell divisions transforming a zygote into an embryo. It also contains the mechanism preventing polyspermy, thereby ensuring successful fertilization and further development.
Retaining extra cytoplasm in a developing oocyte boosts its survival chances by supplying the zygote, formed post-fertilization, with essential nutrients needed during the period up to implantation into the uterus. This is crucial as the sperm solely contributes DNA and not cytoplasm at fertilization. Accordingly, the cytoplasm and all of the cytoplasmic organelles in the developing embryo, including mitochondria, are maternally sourced. Mitochondrial DNA is maternally inherited and critical for tracing biological lineage.
Upon fertilization, the larger amount of cytoplasm also provides energy and raw materials for the rapid cell divisions that take place in the zygote during its journey to the uterus. This process transforms the zygote from a single cell into a tightly packed sphere of cells referred to as a morula which consequently evolves into a blastocyst. The blastocyst implants in the uterine wall and further develops into an embryo.
Furthermore, the oocyte's cytoplasm possesses a mechanism to prevent polyspermy, the penetration of more than one sperm. Polyspermy leads to a triploid organism with three sets of chromosomes which is incompatible with life. Therefore, the cytoplasm plays a crucial role in ensuring successful fertilization and development of the embryo, making enhancing cytoplasmic retention essential for the survival of the developing oocyte.
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Answer:
its C, The child may or may not have the disease because a recessive gene from each parent may be passed on.
Explanation:
Answer:
peroxisome
Explanation:
Adrenoleukodystrophy (ALD) is a X linked recessive disorder. In this disorder, fatty acid beta oxidation occurring in peroxisomes is impaired which leads to the accumulation of long chain fatty acid in cells.
The disease is caused by mutation in gene ABCD1 which codes for peroxisomal membrane transporter. This transporter transports the long chain fatty acids inside peroxisome for degradation hence problem with it interferes with the degradation process leading to build up of fatty acids inside cells.
The main damage is caused to the myelin sheath of the nerves which leads to hyperactivity and seizures. Other symptoms are speaking and listening problems, muscle stiffness, sexual dysfunction etc.